Development of a high-affinity anti-ROR1 variable region for broad anti-cancer immunotherapy
Journal Title
Molecular Therapy
Publication Type
Online publication before print
Abstract
Receptor tyrosine kinase-like orphan receptor 1 (ROR1) is an emerging target in cancer immunotherapy, recognized for its consistent and elevated expression across several epithelial tumors, including triple-negative breast cancer (TNBC). TNBC is an aggressive and difficult-to-treat cancer, with limited effective therapeutic options currently available. Therapeutic approaches centered on targeting ROR1 have therefore become increasingly popular, with ROR1 chimeric antigen receptor (CAR) T cells currently in clinical trials to treat TNBC patients. While ROR1-targeting therapies have shown promising preclinical results, single arm treatment has often shown low efficacy as well as off-target toxicity. Natural killer (NK) cell-based immunotherapies, such as antibody-dependent cell cytotoxicity-inducing monoclonal antibodies and CAR NK cells, have also been shown to induce cancer cell cytotoxicity; however, with less toxicity compared with CAR T cells. Here, we developed and characterized a phage-derived single-chain fragment variable (scFv) against a highly specific ROR1 region and generated scFv-derived chimeric monoclonal antibodies and anti-ROR1-CAR NK cells, which show anti-cancer efficacy against TNBC cells. Additionally, we found TGF-β inhibition using either small-molecule inhibitors or CRISPR-Cas9-edited NK cells could further enhance ROR1-targeting therapy persistence and efficacy in controlling TNBC tumor growth.
Keywords
Adcc; CAR NK cells; CRISPR/Cas9; Frizzled-Kringle domain; TGFBR2 knockout; antibody drug conjugate; natural killer cells; phage display; scFv; triple negative breast cancer
Department(s)
Laboratory Research
Terms of Use/Rights Notice
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Creation Date: 2026-01-27 11:48:49
Last Modified: 2026-01-27 11:48:55
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