Hit-to-lead optimization of novel phenyl imidazole carboxamides that are active against Leishmania donovani
Journal Title
European Journal of Medicinal Chemistry
Publication Type
Research article
Abstract
Visceral leishmaniasis is a potentially fatal disease caused by the parasitic protists, Leishmania donovani and L. infantum. Current treatments remain unsuitable due to cost, the need for hospitalization, variable efficacy against different species, toxicity and emerging resistance. Herein, we report the SAR exploration of the novel hit 4-Fluoro-N-(5-(4-methoxyphenyl)-1-methyl-1H-imidazole-2-yl)benzamide [1] previously identified from a high throughput screen against Trypanosoma brucei, Trypanosoma cruzi and Leishmania donovani. An extensive and informative set of analogues were synthesized incorporating key modifications around the scaffold resulting in improved potency, whilst the majority of compounds maintained low cytotoxicity against human THP-1 macrophages that are target cells for these pathogens. New lead compounds identified within this study also maintained desirable physicochemical properties, improved metabolic stability in vitro and displayed no significant mitotoxicity against HepG2 cell lines. This compound class warrants continued investigation towards development as a novel treatment for Visceral Leishmaniasis.
Publisher
Elsevier
Keywords
*Antiprotozoal Agents/chemistry; Humans; Imidazoles/therapeutic use; *Leishmania donovani; *Leishmaniasis, Visceral/drug therapy; *Trypanosoma cruzi; Early hit-to-lead SAR exploration; L. donovani inhibitors; Neglected tropical disease; Phenyl imidazole carboxamides; Visceral leishmaniasis
Department(s)
Laboratory Research
PubMed ID
35810535
Terms of Use/Rights Notice
Refer to copyright notice on published article.


Creation Date: 2025-01-31 05:10:57
Last Modified: 2025-01-31 05:11:49

© 2025 The Walter and Eliza Hall Institute of Medical Research. Access to this website is subject to our Privacy Policy and Terms of Use

An error has occurred. This application may no longer respond until reloaded. Reload 🗙